modelCediranib

Diagram of Cediranib

Extends from Pharmacolibrary.Drugs.ATC.L.L01EK02.

Information

name:Cediranib
ATC code:L01EK02
route:oral
compartments:1
dosage:45mg
volume of distribution:68L
clearance:32L/h
other parameters in model implementation

Cediranib is an orally available small-molecule tyrosine kinase inhibitor that selectively inhibits vascular endothelial growth factor (VEGF) receptors. It was developed primarily for its anti-angiogenic properties, targeting tumor blood vessel growth in various cancers. Cediranib has been investigated in clinical trials for malignancies such as ovarian, lung, and colorectal cancers, but as of 2024, it is not approved for routine clinical use by major regulatory agencies.

Pharmacokinetics

Pharmacokinetic parameters in adult cancer patients (various solid tumors) after single and multiple oral doses of cediranib.

References

  1. Al-Huniti, N, et al., & Li, J (2018). Population exposure-safety analysis of cediranib for Phase I and II studies in patients with cancer. British journal of clinical pharmacology 84(4) 726–737. DOI:10.1111/bcp.13495 PUBMED:https://pubmed.ncbi.nlm.nih.gov/29274100

  2. Li, J, et al., & Masson, E (2017). Population pharmacokinetic and exposure simulation analysis for cediranib (AZD2171) in pooled Phase I/II studies in patients with cancer. British journal of clinical pharmacology 83(8) 1723–1733. DOI:10.1111/bcp.13266 PUBMED:https://pubmed.ncbi.nlm.nih.gov/28213941

  3. van Herpen, CM, et al., & de Jonge, MJ (2013). Pharmacokinetics and tolerability of cediranib, a potent VEGF signalling inhibitor, in cancer patients with hepatic impairment. Anti-cancer drugs 24(2) 204–211. DOI:10.1097/CAD.0b013e32835bd1d2 PUBMED:https://pubmed.ncbi.nlm.nih.gov/23197081

Parameters

TypeNameDefaultDescription
Modelica.Units.SI.Massweight (from PK_1C)75patient weight (kg)
Modelica.Units.SI.SpecificVolumeVdPerKg (from PK_1C)0.9Volume of distribution (L/kg)
Modelica.Units.SI.MassFractionF (from PK_1C)0.8bioavailiability (0-1)
Pharmacolibrary.Types.ClearanceCl (from PK_1C)20clearance
Modelica.Units.SI.TimeadminTime (from PK_1C)60first administration time (s)
Modelica.Units.SI.TimeadminDuration (from PK_1C)600administration duration (s)
Modelica.Units.SI.TimeadminPeriod (from PK_1C)8*60*60period of administration (default 8 hours)(s)
Pharmacolibrary.Types.MassadminMass (from PK_1C)1000administration mass (mg)
IntegeradminCount (from PK_1C)8number of dose administered (1)
Pharmacolibrary.Types.VolumeVd (from PK_1C)VdPerKg*weightVolume of distribution (m3)
Pharmacolibrary.Types.MassConcentrationCmin (from PK_1C)0.004minimal therapeutic range
Pharmacolibrary.Types.MassConcentrationCmax (from PK_1C)0.008minimal therapeutic range
Pharmacolibrary.Types.MassConcentrationCtox_peak (from PK_1C)0.012toxicity peak level
Pharmacolibrary.Types.MassConcentrationCtox_trough (from PK_1C)0.006toxicity trough level
Pharmacolibrary.Types.TransferRateka (from PK_1C_enteral)0.016666666666666666first order absorption rate
Modelica.Units.SI.TimeTlag (from PK_1C_enteral)600delay between oral administration and absorption (default 10min)

Connectors

TypeNameDefaultDescription
Types.ConcentrationOutputC_central (from PK_1C)
Interfaces.ConcentrationPort_bcentralCPort (from PK_1C)

Components

TypeNameDefaultDescription
Pharmacokinetic.NoPerfusedTissueCompartmentcentral (from PK_1C)
Pharmacokinetic.ClearanceDrivenEliminationelim (from PK_1C)
Sources.PeriodicDoseperiodicDose (from PK_1C)
Modelica.Units.SI.Timet1_2 (from PK_1C)elimination half-life

Revisions

  • 06/2025 Tomas Kulhanek, generated model from data extracted from PUBMED, DrugBank and LLM(GPT4.1)