modelNivolumab

Diagram of Nivolumab

Extends from Pharmacolibrary.Drugs.ATC.L.L01FF01.

Information

name:Nivolumab
ATC code:L01FF01
route:intravenous
compartments:2
dosage:3mg
volume of distribution:8.0L
clearance:0.28L/day
other parameters in model implementation

Nivolumab is a fully human IgG4 monoclonal antibody that inhibits the programmed death-1 (PD-1) immune checkpoint pathway. It is approved for the treatment of various cancers, including metastatic melanoma, non-small cell lung cancer, renal cell carcinoma, and others. Nivolumab enhances anti-tumor immune responses by blocking PD-1 receptor from binding to its ligands, PD-L1 and PD-L2.

Pharmacokinetics

Pharmacokinetic parameters were obtained from studies in adult patients with cancer, both male and female, across different tumor types. Data primarily reflect intravenous administration in clinical trial populations.

References

  1. Albiges, L, et al., & George, S (2025). Subcutaneous versus intravenous nivolumab for renal cell carcinoma. Annals of oncology : official journal of the European Society for Medical Oncology 36(1) 99–107. DOI:10.1016/j.annonc.2024.09.002 PUBMED:https://pubmed.ncbi.nlm.nih.gov/39288844

  2. Osawa, M, et al., & Hruska, MW (2019). Population pharmacokinetics analysis of nivolumab in Asian and non-Asian patients with gastric and gastro-esophageal junction cancers. Cancer chemotherapy and pharmacology 83(4) 705–715. DOI:10.1007/s00280-019-03771-z PUBMED:https://pubmed.ncbi.nlm.nih.gov/30666395

  3. Wang, W, et al., & Yang, G (2024). Pharmacokinetics, Safety, and Immunogenicity of a Biosimilar of Nivolumab (LY01015): A Randomized, Double-Blind, Parallel-Controlled Phase I Clinical Trial in Healthy Chinese Male Subjects. BioDrugs : clinical immunotherapeutics, biopharmaceuticals and gene therapy 38(6) 855–865. DOI:10.1007/s40259-024-00679-w PUBMED:https://pubmed.ncbi.nlm.nih.gov/39317850

Parameters

TypeNameDefaultDescription
Modelica.Units.SI.Massweight (from PK_1C)75patient weight (kg)
Modelica.Units.SI.SpecificVolumeVdPerKg (from PK_1C)0.9Volume of distribution (L/kg)
Modelica.Units.SI.MassFractionF (from PK_1C)0.8bioavailiability (0-1)
Pharmacolibrary.Types.ClearanceCl (from PK_1C)20clearance
Modelica.Units.SI.TimeadminTime (from PK_1C)60first administration time (s)
Modelica.Units.SI.TimeadminDuration (from PK_1C)600administration duration (s)
Modelica.Units.SI.TimeadminPeriod (from PK_1C)8*60*60period of administration (default 8 hours)(s)
Pharmacolibrary.Types.MassadminMass (from PK_1C)1000administration mass (mg)
IntegeradminCount (from PK_1C)8number of dose administered (1)
Pharmacolibrary.Types.VolumeVd (from PK_1C)VdPerKg*weightVolume of distribution (m3)
Pharmacolibrary.Types.MassConcentrationCmin (from PK_1C)0.004minimal therapeutic range
Pharmacolibrary.Types.MassConcentrationCmax (from PK_1C)0.008minimal therapeutic range
Pharmacolibrary.Types.MassConcentrationCtox_peak (from PK_1C)0.012toxicity peak level
Pharmacolibrary.Types.MassConcentrationCtox_trough (from PK_1C)0.006toxicity trough level
Pharmacolibrary.Types.VolumeVdp (from PK_2C)VdpPerKg*weightVolume of distribution (m3)
Modelica.Units.SI.SpecificVolumeVdpPerKg (from PK_2C)0.9Volume of distribution peripheral(l/kg)
Pharmacolibrary.Types.Clearancek12 (from PK_2C)1intercompartmental C-P clearance
Pharmacolibrary.Types.Clearancek21 (from PK_2C)1intercompartmental P-C clearance

Connectors

TypeNameDefaultDescription
Types.ConcentrationOutputC_central (from PK_1C)
Interfaces.ConcentrationPort_bcentralCPort (from PK_1C)
Interfaces.ConcentrationPort_bperipheralCPort (from PK_2C)
Pharmacolibrary.Types.ConcentrationOutputC_peripheral1 (from PK_2C)

Components

TypeNameDefaultDescription
Pharmacokinetic.NoPerfusedTissueCompartmentcentral (from PK_1C)
Pharmacokinetic.ClearanceDrivenEliminationelim (from PK_1C)
Sources.PeriodicDoseperiodicDose (from PK_1C)
Modelica.Units.SI.Timet1_2 (from PK_1C)elimination half-life
Pharmacolibrary.Pharmacokinetic.TransferFirstOrderNonSymtransfer (from PK_2C)
Pharmacolibrary.Pharmacokinetic.NoPerfusedTissueCompartmentperipheral (from PK_2C)

Revisions

  • 06/2025 Tomas Kulhanek, generated model from data extracted from PUBMED, DrugBank and LLM(GPT4.1)